Marcus aka Gregory Maidman
1 min readApr 7, 2021

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I am not talking about a triggering an immune response. I am talking about designing a site receptor inhibitor to bind to the cell entry site and thus impair the virus abiity to infect and multiply. For example ACE2 inhibitors already exist but the problem with any site blocker is weak affinity. The Spike protein would likely have greater affinity. I am not suggesting it as an alternative to a vaccine but rather as a prophilaxis supplement. The fact is they still have no idea whether or not the vaccines fight the bug or fight the symptoms. I am not an epidemiologist but I understand basic math and logic. The studies are grossly deceptive. Using rough numbers, 21,000 people got the placebo and 140 were counted as infected. That's 0.7%. That means according to the study one has a far better chance of not getting sick simply by being in the study, which is of course nonsense and points out the fact that the study protocol did not include testing unless someone reported symptoms. I wrote many articles on the fallacies of the studies. Here is one. https://medium.com/grab-a-slice/pfizer-and-moderna-vaccines-95-efficacy-are-opinions-which-fail-the-federal-standards-for-d271e2f2a3b0. I would not be surprised if the mRNA vaccines, which hijack our own cells to produce the spike protein to trigger in effect an auto-immune response, are actually also blocking the receptor sites

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Marcus aka Gregory Maidman

Living 17,043rd human life. I am Marcus (universal name) or you may call me Greg; a deep thinker; an explorer of ideas and the mind.